In August 2006, the American Diabetes Association journal Diabetes reported in an article published on the letter and significantly Yonsei University of North Carolina at Chapel Hill, Maeda results, to give α-lipoic acid in diabetic patients can not be induced increases in cholesterol, blood atherosclerotic disease and poor health, or the reasons.

α-lipoic acid is a potent antioxidant nutrient that is used to treat diabetic neuropathy, however, the impact of diabetes, cardiovascular disease has not been fully evaluated.

Drs Yi and Maeda, which with the United Nations Command Department of Anatomy Pathology and Laboratory Medicine, induced diabetes in a group of three months old-deficient mice and apolipoprotein management STZ (STZ), while the control group in the buffer solution.

After 5 weeks of normal diet, diabetes, and non-diabetic mice were given high-fat diet and increased Alpha – Alpha Lipoic or the same diet – Lipoic.

Body weight, blood sugar and cholesterol levels, to determine the start of the study, every 4 weeks until 20 weeks later.

The levels of erythrocyte glutathione and oxidative stress markers measured in the study, when the animal for the study of development of atherosclerotic plaques, and for the treatment of the effects of pancreatic islets βcells.

After 20 weeks, both diabetic and non-diabetic animals, the end of the added α-lipoic acid in oxidative stress and higher levels of erythrocyte glutathione in mice compared to lower voting, did not receive the compound.

Although the diabetic mice that did not receive a lot of α-lipoic acid and the disease began to appear signs of drowsiness caused by the disease for three months, who received α-lipoic acid diabetic mice appeared healthy throughout the study period.

“It’s worth noting that,” the authors write, “α-lipoic acid in the complete cessation of increase in plasma total cholesterol, atherosclerotic lesions, and general health caused by diabetes.”

Although the hyperglycemia induced by diabetes mellitus after the first gradually decreased in mice that received and did not receive 20 weeks of α-lipoic acid, the levels of blood glucose in mice with a low supplement.

The researchers put the blame on some protection or regeneration of β cells, pancreatic alpha – lipoic acid, as found that mice that received the compounds are more insulin-producing cells.

In short, “Our research shows that dietary α-lipoic acid completely protect 20 Zhou apoplipoprotein electronic aortic atherosclerosis induced by STZ-induced diabetic mice enhanced the negative,” the authors conclude. “Our results suggest that dietary α-lipoic acid is to reduce cardiovascular complications of diabetes, a promising agent.

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